Dr. Zhe Zhang is the Associate professor at Infection and
She obtained her B.S. degree from the department of microbiology at
After she joined
Webpage@ School of medicine: http://www.cmm.zju.edu.cn/chinese/scholar.php?uid=644
Xuai Lin, Aihua Sun, Ping Ruan, Zhe Zhang, Jie Yan. Characterization of Conserved Combined T and B Cell Epitopes in Leptospira interrogans Major Outer Membrane Proteins OmpL1 and LipL41. BMC Microbiol. 2011, Jan 26;11(1):21.
Zhe Zhang, Thomas B. Clarke, and Jeffrey N. Weiser. Cellular effectors mediating Th17-dependent clearance of pneumococcal colonization in mice. J Clin Invest, 2009, 119:1899-1909. *Science News (ScienceDaily, June 17, 2009): Clearing pneumococcal bacteria from the upper airways.
Zhe Zhang, William Reenstra, Daniel J. Weiner, Jean-Pierre Louboutin, and James M. Wilson. The p38 mitogen-activated protein kinase signaling pathway is coupled to Toll-like receptor 5 to mediate gene regulation in response to Pseudomonas aeruginosa infection in human airway epithelial cells. Infect Immun, 2007, 75:5985-5992.
Zhe Zhang, Jean-Pierre Louboutin, Daniel J. Weiner, Joanna B. Goldberg, and James M. Wilson. Human airway epithelial cells sense Pseudomonas aeruginosa infection via recognition of flagellin by Toll-like receptor 5. Infect Immun, 2005, 73: 7151-7160.
Wen-Bo Zhang, Zhe Zhang (co-first author), Yan-Xiang Ni, Ya-Lan Wu and
Xiaorong Wang, Zhe Zhang, Jean-Pierre Louboutin, Christian Moser, Daniel J. Weiner, and James M. Wilson. Airway epithelia regulate expression of human β-defensin 2 through toll-like receptor 2. FASEB J, 2003, 17:1727-1729.
Zhu-Bin Lei, Zhe Zhang, Qing Jing, Yong-Wen Qin, Gang Pei, Beng-Zhen Cao, Xiao-Yan Li. OxLDL upregulates CXCR2 expression in monocytes via scavenger receptors and activation of p38 mitogen-activated protein kinase. Cardiovascular Research, 2002, 53: 524–532.
Zhe Zhang, Shun-Mei Xin, Guo-Xiang Wu, Wen-Bo Zhang, Lan Ma, and
G 蛋白偶联受体介导的MAPK的激活.《细胞生物学动态》，1999第3卷: 55-62. 张喆，裴钢.
Li-Guang Lou, Zhe Zhang (co-first author), Lan Ma, and
Guo-huang Fan, Jing Zhao, Ya-Lan Wu, Li-Guang Lou, Zhe Zhang, Qing Jing, Lan Ma, and Gang Pei. N-methyl-D-aspartate attenuates opioid receptor-mediated G protein activation and this process involves protein kinase C. Molecular Pharmacology, 1998, 53:684–690.
Zhi-Jie Cheng, Guo-Huang Fan, Jian Zhao, Zhe Zhang, Ya-Lan Wu, Li-Zhen Jiang, Ye Zhu, Gang Pei, and Lan Ma. Endogenous opioid receptor-like receptor in human neuroblastoma SK-N-SH cells: activation inhibitory G protein and homologous desensitization. NeuroReport, 1997, 8:1913-1918.
ORL1受体在SK-N-SH细胞中的功能性表达及其跨膜信号转导.山东大学学报（自然科学版），1997，第32卷，第2期: 231-236. 张喆，蔡迎春，尹德领，蒋丽珍，裴钢.
A positive feedback interaction promotes rapid formation of BALT in response to pulmonary mycobacteria infection in BCG-vaccinated mice. Zhe Zhang. BIT’s 2nd Annual World Congress of Microbes (Guangzhou, China, 2012), WCM conference Journal (2012）p 316.
TLR and Myd88 signaling pathway is required for the full induction of host adaptive immune responses to adenoviral vectors and transgenes. (9th Annual Meeting of American Society of Gene Therapy,
Acute-Phase toxicity following systemic administration of non-Human primate derived adenoviral vectors to mice. (9th Annual Meeting of American Society of Gene Therapy,
TLR and MyD88 signaling mediates innate immune responses to adenoviral vectors in mouse. (9th Annual Meeting of American Society of Gene Therapy,
Role of MyD88 signaling pathway in the host immune responses in adenoviral vector-mediated gene therapy. (8th Annual Meeting of American Society of Gene Therapy,
Differential roles of polymorphonuclear leukocytes, alveolar macrophages and airway epithelial cells and their coordination by TLR4-MyD88 signaling pathway in mouse pulmonary antibacterial defense to Heamophilus influenzae. (19th Annual North American Cystic Fibrosis Conference,
Dissecting the cellular and molecular components of mouse pulmonary defense against Pseudomonas aeruginosa. (19th Annual North American Cystic Fibrosis Conference,
Role of TLR5-p38 MAPK signaling pathway in the pulmonary innate immune response to Pseudomonas aeruginosa. (19th Annual North American Cystic Fibrosis Conference,
Role of TLR5-p38 MAPK signaling pathway in the pulmonary innate immune response to Pseudomonas aeruginosa. (18th Annual North American Cystic Fibrosis Conference,
Differential Role of Toll-Like Receptors and Alveolar Macrophages in the Clearance of Heamophilus influenzae and Pseudomonas aeruginosa in mouse lung. (18th Annual North American Cystic Fibrosis Conference,
Pseudomonas areuginosa upregulates gene expression through TLR5-mediated p38 MAPK signaling pathway in human airway epithelial cells. (17th Annual North American Cystic Fibrosis Conference,
The role of Toll-like receptors and neutrophilic infiltration in clearece of Haemophilus influenzae and Pseudomonas areuginosa. (17th Annual North American Cystic Fibrosis Conference,
Pulmonary innate immune response to Pseudomonas areuginosa is mediated by Toll-like receptors. (16th Annual North American Cystic Fibrosis Conference,
Prevention and treatment of respiratory tract infectious diseases.